spinal stimulator dorsal column myelopathy

Spinal dural arteriovenous fistulas: MR and myelographic findings. Gilbertson JR, Miller GM, Goldman MS, Marsh WR. Complications of lumbar puncture followed by anticoagulation. Idiopathic spontaneous intraspinal intramedullary hemorrhage: a report of two cases and literature review. Characteristics of spontaneous spinal cord infarction and proposed diagnostic criteria. Zalewski NL, Rabinstein AA, Krecke KN, Brown RD Jr, Wijdicks EFM, Weinshenker BG, et al. Resolution of syringomyelia and Chiari I malformation by ventriculoatrial shunting in a patient with pseudotumor cerebri and a lumboperitoneal shunt. Chiari I malformation with syrinx: an unexpected diagnosis in the emergency department. Prattico F, Perfetti P, Gabrieli A, Longo D, Caroselli C, Ricci G. Symptomatic atlantoaxial instability in Down syndrome. Nader-Sepahi A, Casey AT, Hayward R, Crockard HA, Thompson D. Rheumatoid arthritis in the cervical spine.

An unusual cause of spontaneous paralysis. Spinal epidural lipomatosis-a brief review. Spinal cord compression with paraplegia in myelofibrosis. Oustwani MB, Kurtides ES, Christ M, Ciric I. Specific pattern of gadolinium enhancement in spondylotic myelopathy. 2010 16(2):120–2.įlanagan EP, Krecke KN, Marsh RW, Giannini C, Keegan BM, Weinshenker BG. Compressive myelopathy mimicking transverse myelitis. Myelin oligodendrocyte glycoprotein-IgG contributes to oligodendrocytopathy in the presence of complement, distinct from astrocytopathy induced by AQP-4 IgG. Clinical, radiologic, and prognostic features of myelitis associated with myelin oligodendrocyte glycoprotein autoantibody. 2012 79(12):1273–7.ĭubey D, Pittock SJ, Krecke KN, Morris PP, Sechi E, Zalewski NL, et al. Myelin-oligodendrocyte glycoprotein antibodies in adults with a neuromyelitis optica phenotype. Kitley J, Woodhall M, Waters P, Leite MI, Devenney E, Craig J, et al. Aquaporin-4-binding autoantibodies in patients with neuromyelitis optica impair glutamate transport by down-regulating EAAT2. Hinson SR, Roemer SF, Lucchinetti CF, Fryer JP, Kryzer TJ, Chamberlain JL, et al. Neuromyelitis optica: passive transfer to rats by human immunoglobulin.

Kinoshita M, Nakatsuji Y, Kimura T, Moriya M, Takata K, Okuno T, et al. Neuromyelitis optica: pathogenicity of patient immunoglobulin in vivo. 2006 63(7):964–8.īradl M, Misu T, Takahashi T, Watanabe M, Mader S, Reindl M, et al. Neuromyelitis optica brain lesions localized at sites of high aquaporin 4 expression. Pittock SJ, Weinshenker BG, Lucchinetti CF, Wingerchuk DM, Corboy JR, Lennon VA. Short myelitis lesions in aquaporin-4-IgG-positive neuromyelitis optica spectrum disorders. 2004 364(9451):2106–12.įlanagan EP, Weinshenker BG, Krecke KN, Lennon VA, Lucchinetti CF, McKeon A, et al. A serum autoantibody marker of neuromyelitis optica: distinction from multiple sclerosis. Lennon VA, Wingerchuk DM, Kryzer TJ, Pittock SJ, Lucchinetti CF, Fujihara K, et al. Spinal cord compression: from laboratory to clinic. Malignant spinal cord compression: prospective study of delays in referral and treatment. Patients whose symptoms continue to progress after initial immunosuppressive treatment may benefit from plasmapheresis and occasionally from biopsy for definitive diagnosis. Empiric high-dose corticosteroid treatment is often indicated in noncompressive myelopathy additional investigations are important to identify patients with potentially relapsing or progressive disorders who may benefit from preventive therapies. The key diagnostic considerations include demyelinating, vascular, inflammatory, infectious, and paraneoplastic disorders. When compression is not detected, an analysis of precise lesion localization, non-neurological clinical features, MRI findings, and serologic studies narrow the differential diagnosis. When cord compression is present, surgical treatment is usually indicated. MRI of the spine is the imaging modality of choice. Patients with signs and symptoms of acute myelopathy require urgent neurologic evaluation focused upon the identification and management of treatable disorders.

Spinal stimulator dorsal column myelopathy